Mucopolysaccharidosis I (MPS I), pronounced mew-ko-pol-ee-sak-ah-ri-doh-sis one, is a rare genetic disorder that affects many body systems and that leads to organ damage. It is caused by a mutation in the gene that makes an enzyme called alpha-L-iduronidase (pronounced al-fa el eye-dur-on-i-dase).
Available immediately from VitalStream's direct sales team, the MediaConsole MPS solution will be demonstrated for the first time during NAB 2005, Las Vegas, NV at VitalStream's Booth #SL1023 and at thePlatform's booth #SL3322.
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MPS I H (also called Hurler syndrome or α-L-iduronidase deficiency), is the most severe of the MPS I subtypes. Developmental delay is evident by the end of the first year, and patients usually stop developing between ages 2 and 4.
Mucopolysaccharidosis I (MPS I) is a rare genetic disorder that affects both physical and mental development and can cause organ damage.
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Mucopolysaccharidosis TYPE II (MPS II) is a serious genetic disorder that primarily affects males. It is one of several related lysosomal storage diseases. MPS interferes with the body’s ability to break down and recycle specific mucopolysaccharides (mew-ko-pol-ee-sak-ah-rides).
Mucopolysaccharidosis type I (MPS I) is a condition that affects many parts of the body. This disorder was once divided into three separate syndromes: Hurler syndrome (MPS I-H), Hurler-Scheie syndrome (MPS I-H/S), and Scheie syndrome (MPS I-S), listed from most to least severe.
MPS I has a wide range of symptoms, and people may experience different degrees of disease progression. Because the signs and symptoms are variable, it affects each individual differently. The list below provides an overview of the signs and symptoms that may occur in individuals with various severities of MPS I.